Basic science research suggests a promising, if surprising, method to protect against anesthesia-induced developmental neurotoxicity: subclinical carbon monoxide (CO) inhalation, according to a new report. Low concentrations of CO limit apoptosis after isoflurane exposure in immature mice, through a mechanism of cytochrome c peroxidase inhibition.

Source: Could Carbon Monoxide Protect Against Anesthesia-Induced Neuroapoptosis?


David Cottle

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